@article{9c74b1588a6649ba893a48ee6df2affb,
title = "Tildacerfont for the treatment of patients with classic congenital adrenal hyperplasia: results from a 12-week phase 2 clinical trial in adults with classic CAH",
author = "Richard Auchus and Deborah Merke and Madu Ivy-Joan and Samer Nakhle and Kyriakie Sarafoglou and Michael Huang and David Moriarty and Chris Barnes and Ron Newfield",
note = "1University of Michigan, USA; 2Eunice Kennedy Shriver National Institute of Child Health and Human Development, USA; 3Diabetes Associates Medical Group, USA; 4Palm Medical Group, USA; 5University of Minnesota Medical School, USA; 6Former employee of Spruce Biosciences, Inc., USA; 7Spruce Biosciences, Inc., USA; 8 University of California San Diego, USA Background Congenital adrenal hyperplasia (CAH) due to 21-hydroxylase deficiency (21-OHD) is an autosomal recessive disorder characterized by insufficient cortisol production resulting in excess adrenocorticotropic hormone (ACTH) and adrenal androgen production.",
year = "2021",
month = may,
day = "15",
doi = "10.1530/ENDOABS.73.PEP1.2",
language = "American English",
volume = "73",
journal = "23rd European Congress of Endocrinology (ECE 2021)",
}